DR. RAJALAKSHMI S.

DR. JUHY CHERIAN, DR.GAINATHI KAMBAM, DR. SIVARAJA GOWTHAMAN ANBARASU

Abstract

Trauma is one of the causes of Orbital Apex Syndrome. Considering a reported incidence of traumatic superior orbital fissure syndrome of only 0.3- 0.8% and of traumatic optic neuropathy of 0.8–6.0%, the incidence of traumatic orbital apex syndrome (coexistence of both conditions) would be expected to be much lower than either alone. Here the authors report two such cases following RTA in young males, wherein fracture of lesser or greater wing of sphenoid impinges upon the contents of superior orbital fissure extending to the optic canal and in-turn compresses the optic nerve causing Traumatic Orbital Apex Syndrome. External ophthalmoplegia, ptosis, proptosis, fixed dilated pupil, anaesthesia of forehead & upper eyelid and relative afferent pupillary defect form the symptom complex of this syndrome. Narrow SOF (less than 1.6mm) is found to be an anatomical risk factor. Steroids were administered in these patients. Surgical decompression in good hands can have a positive outcome.